Kamis, 26 Januari 2012

CHOLESTEROL 10 - Atherosclerosis 6



PULIH MARI BALI WUTUH PURNA WALUYA JATI

Daftar Jamu Godog Kendhil Kencana >>>Jamu Godog Penurun Cholesterol – LEMAK NORMAL

Cross-sectional diagrams of human blood vessels showing a normal, healthy artery and a narrowed, atherosclerotic artery.
Common and specific pathogenetic features of aortic stenosis and atherosclerosis.
Mechanisms of Vessel Wall Neovascularization in Atherosclerosis
Increased proangiogenic activity, which may be accompanied by reduced endogenous inhibition of angiogenesis, results in vasa vasorum proliferation and intimal neovascularization. FGF = fibroblast growth factors; HGF = hepatocyte growth factor; PDGF = platelet-derived growth factor; SMC = smooth muscle cell; VEGF= vascular endothelial growth factor.
Copyrighted and used with permission of Mayo Foundation for Medical Education and Research.
Cellular interactions in the development and progression of atherosclerosis. VSMC, vascular smooth muscle cells.
Thrombotic complication of atherosclerosis.
Ultimately, inflammatory mediators can inhibit collagen synthesis and evoke the expression of collagenases by macrophage foam cells within the intima. 
This imbalance diminishes the collagen content of the fibrous cap, rendering it weak and rupture-prone. 
In parallel, crosstalk between T lymphocytes and other cell types present within lesions heightens the expression of the potent procoagulant tissue factor. 
Thus, when the fibrous cap ruptures, as illustrated in this diagram, tissue factor induced 
by inflammatory signaling triggers the thrombus that causes most acute complications of atherosclerosis. 
Clinically, this may translate into an acute coronary syndrome.

Daftar Jamu Godog Kendhil Kencana >>>Jamu Godog Penurun Cholesterol – LEMAK NORMAL

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